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Biblioteca (s) : |
INIA Tacuarembó. |
Fecha : |
29/04/2015 |
Actualizado : |
16/09/2016 |
Tipo de producción científica : |
Trabajos en Congresos/Conferencias |
Autor : |
DO CARMO, M.; CARDOZO, G.; SOCA, P.M. |
Afiliación : |
MARTIN DO CARMO CORUJO, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; GERONIMO AGUSTIN CARDOZO CABANELAS, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; PABLO M. SOCA. |
Título : |
Measurement of campos rangelands herbage mass by simple methods. [Resumen]. |
Fecha de publicación : |
2015 |
Fuente / Imprenta : |
ANNUAL MEETING OF THE SOCIETY FOR RANGE MANAGEMENT, SRM, 68., Sacramento, USA, 2015. |
Idioma : |
Inglés |
Contenido : |
Accurate and precise estimation of herbage mass is essential for livestock management, because herbage allowance depends on it estimation. Difficulties in estimation arise because of great variability in herbage mass in grassland and rangeland situations. Measuring yield directly by cutting is costly and destructive; but herbage height is highly correlated with herbage mass, thus could be a substitute of the cutting. Based on measurement of herbage mass in a large experiment (EEBR) and in four farms across Uruguay (MICN), we analyzed the relationship between herbage height and dry weight of the quadrat in kg/ha. Analysis was performed
with PROC MIXED and PROC CORR of SAS. On the other hand, the sample size to be taken in each paddock is of great importance and was estimated from the data population described above by resampling and estimating the coefficient of variation (CV) between means. A CV of 10% was defined as the limit to consider the estimation precise enough. The general relationship was y = 224 (±36) + x * 339 (±4), R2 = 0.84. Sample size to maintain the average with a CV error below 10% varied with mean, because it change the variance, however as a general rule, with a sample size between 60 to 100 only one estimation from 198, was above 10% error of the mean. We conclude that estimations based on herbage height are accurate enough (R2 = 0.84) to estimate the herbage mass and a sample size from
60 to 100, give as confidence to estimate herbage mass with precision in paddocks from 5 to 190 ha. That methodology should be extended to Campos rangeland manager for decision making to control grazing intensity. MenosAccurate and precise estimation of herbage mass is essential for livestock management, because herbage allowance depends on it estimation. Difficulties in estimation arise because of great variability in herbage mass in grassland and rangeland situations. Measuring yield directly by cutting is costly and destructive; but herbage height is highly correlated with herbage mass, thus could be a substitute of the cutting. Based on measurement of herbage mass in a large experiment (EEBR) and in four farms across Uruguay (MICN), we analyzed the relationship between herbage height and dry weight of the quadrat in kg/ha. Analysis was performed
with PROC MIXED and PROC CORR of SAS. On the other hand, the sample size to be taken in each paddock is of great importance and was estimated from the data population described above by resampling and estimating the coefficient of variation (CV) between means. A CV of 10% was defined as the limit to consider the estimation precise enough. The general relationship was y = 224 (±36) + x * 339 (±4), R2 = 0.84. Sample size to maintain the average with a CV error below 10% varied with mean, because it change the variance, however as a general rule, with a sample size between 60 to 100 only one estimation from 198, was above 10% error of the mean. We conclude that estimations based on herbage height are accurate enough (R2 = 0.84) to estimate the herbage mass and a sample size from
60 to 100, give as confidence to estimate herbage mass with precisi... Presentar Todo |
Thesagro : |
FORRAJES; PASTURAS. |
Asunto categoría : |
A50 Investigación agraria |
URL : |
http://www.ainfo.inia.uy/digital/bitstream/item/4400/1/DO-CARMO-2015-AINFO.pdf
http://www.rangelands.org/events/
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Marc : |
LEADER 02135nam a2200157 a 4500 001 1052580 005 2016-09-16 008 2015 bl uuuu u01u1 u #d 100 1 $aDO CARMO, M. 245 $aMeasurement of campos rangelands herbage mass by simple methods. [Resumen].$h[electronic resource] 260 $aANNUAL MEETING OF THE SOCIETY FOR RANGE MANAGEMENT, SRM, 68., Sacramento, USA$c2015 520 $aAccurate and precise estimation of herbage mass is essential for livestock management, because herbage allowance depends on it estimation. Difficulties in estimation arise because of great variability in herbage mass in grassland and rangeland situations. Measuring yield directly by cutting is costly and destructive; but herbage height is highly correlated with herbage mass, thus could be a substitute of the cutting. Based on measurement of herbage mass in a large experiment (EEBR) and in four farms across Uruguay (MICN), we analyzed the relationship between herbage height and dry weight of the quadrat in kg/ha. Analysis was performed with PROC MIXED and PROC CORR of SAS. On the other hand, the sample size to be taken in each paddock is of great importance and was estimated from the data population described above by resampling and estimating the coefficient of variation (CV) between means. A CV of 10% was defined as the limit to consider the estimation precise enough. The general relationship was y = 224 (±36) + x * 339 (±4), R2 = 0.84. Sample size to maintain the average with a CV error below 10% varied with mean, because it change the variance, however as a general rule, with a sample size between 60 to 100 only one estimation from 198, was above 10% error of the mean. We conclude that estimations based on herbage height are accurate enough (R2 = 0.84) to estimate the herbage mass and a sample size from 60 to 100, give as confidence to estimate herbage mass with precision in paddocks from 5 to 190 ha. That methodology should be extended to Campos rangeland manager for decision making to control grazing intensity. 650 $aFORRAJES 650 $aPASTURAS 700 1 $aCARDOZO, G. 700 1 $aSOCA, P.M.
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INIA Tacuarembó (TBO) |
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Registro completo
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Biblioteca (s) : |
INIA La Estanzuela. |
Fecha actual : |
02/03/2017 |
Actualizado : |
07/03/2017 |
Autor : |
GARCIA, J.P; GIANNITTI, F.; FINNIE, J.W.; MANAVIS, J. .; BEINGESSER, J.; ADAMS ,V.; ROOD, J.I.; UZAL, F.A. |
Afiliación : |
FEDERICO GIANNITTI, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay. |
Título : |
Comparative Neuropathology of Ovine Enterotoxemia Produced by Clostridium perfringens Type D Wild-Type Strain CN1020 and Its Genetically Modified Derivatives. |
Fecha de publicación : |
2015 |
Fuente / Imprenta : |
Veterinary Pathology, 2015, v.52.n.6. p.1250-1253, 2015. |
Idioma : |
Inglés |
Contenido : |
Abstract
Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated with anti?amyloid precursor protein immunohistochemistry. Perivascular accumulation of macrophages/microglia with intracytoplasmic albumin globules was also observed in these animals. This study demonstrates that ETX is responsible for the major cerebrovascular changes in C. perfringens type D?induced disease. MenosAbstract
Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated wi... Presentar Todo |
Thesagro : |
ENFERMEDADES DE LOS ANIMALES. |
Asunto categoría : |
-- |
Marc : |
LEADER 02512naa a2200217 a 4500 001 1056750 005 2017-03-07 008 2015 bl uuuu u00u1 u #d 100 1 $aGARCIA, J.P 245 $aComparative Neuropathology of Ovine Enterotoxemia Produced by Clostridium perfringens Type D Wild-Type Strain CN1020 and Its Genetically Modified Derivatives.$h[electronic resource] 260 $c2015 520 $aAbstract Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated with anti?amyloid precursor protein immunohistochemistry. Perivascular accumulation of macrophages/microglia with intracytoplasmic albumin globules was also observed in these animals. This study demonstrates that ETX is responsible for the major cerebrovascular changes in C. perfringens type D?induced disease. 650 $aENFERMEDADES DE LOS ANIMALES 700 1 $aGIANNITTI, F. 700 1 $aFINNIE, J.W. 700 1 $aMANAVIS, J. . 700 1 $aBEINGESSER, J. 700 1 $aADAMS ,V. 700 1 $aROOD, J.I. 700 1 $aUZAL, F.A. 773 $tVeterinary Pathology, 2015$gv.52.n.6. p.1250-1253, 2015.
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